TNBC risk increases with the defect of estrogen signnaling. https://t.co/aYUNuNJ1bG
The grade of defect in metabolic and hormonal equilibrium is directly associated with TNBC risk https://t.co/0DauctL2gi
The stronger the defect of estrogen signaling the higher is the risk for TNBC development. https://t.co/jYoAUXqaab
The better the estrogen supply of a patient, the lower is the possibility for TNBC development. https://t.co/FJZUrSJgVP
Tumor suppressing effect of extreme or the liberating effect of defective estrogen signaling (multiparity and nulli https://t.co/QSoUmyo9va
Both deficient estrogen exposure and decreased reactivity of estrogen receptors (estrogen resistance) may increase https://t.co/D1kxt8K1Pa
The weaker the estrogen signaling, the higher is the risk for poorly differetiated TNBC development. https://t.co/YkWAJMTKXi
In African American women, increased risk for TNBC may be attributed to the hormonal dysfunctions caused by the rel https://t.co/gKykv3qgMe
The stronger the defect of estrogen signaling in a patient, the higher is the risk for lowly differentiated TNBC. https://t.co/A87tNmIqAm
ER- breast cancer is not a distinct entity being separated from ER+ ones, but rather a poorly differentiated tumor https://t.co/Oks6qUk9AY
The stronger the estrogen signaling in premenopausal women, the better is the defense against ER+ cancers, while th https://t.co/XIKJdhcf77
In premenopausal women, cyclic estrogen synthesis is a stronger defense against ER+ breast cancer as compared with https://t.co/AhJqC6Dzso
TNBC risk is not higher in cycling premenopausal women, but rather they have stronger defence against ER+ tumors an https://t.co/bftyBfoqQg
TNBC risk in women is defined by the defective E2 signaling both in premenopausal and postmenopausal women. https://t.co/BabEtwQFLy
Insulin resistance and deficient estrogen signaling defines the risk for TNBC development in both pre- and postmeno https://t.co/lWxzQ3hzIT
Both deficient estrogen exposure and decreased reactivity of estrogen receptors (estrogen resistance) may increase https://t.co/Raa3ZxjmFn
The stronger the defect of estrogen signaling, the higher is the risk for ER-negative breast cancer. https://t.co/5jOAvE0RVW
The stronger the estrogen signaling of patients, the better is the defense against TNBC development. https://t.co/baPKnWzmCl
Health disparities of African-American women may be attributed to incongruence between their dark skin and low ligh https://t.co/hfULksRqpi
The grade of defect in metabolic and hormonal equilibrium is directly associated with TNBC risk for both premenopau https://t.co/SHJfjZSu5q
The grade of defect in glucose uptake and ER signaling is directly associated with TNBC risk. https://t.co/HxyAIV2FTY
The higher the ER expression of tumor cells, the stronger the killing capacity of estrogen exposure. https://t.co/zbqND8fgSi
The grade of defect in metabolic and hormonal equilibrium is directly associated with TNBC risk. https://t.co/eyNhZAn09p
Higher TNBC risk in young women as compared with older cases is a statistical mistake. https://t.co/b3KPkSVKXM
The grade of defect in metabolic and hormonal equilibrium is directly associated with TNBC risk for both premenopau https://t.co/CrDtfAeSaX
Low light exposure of black skinned American women causes dysparity in poorly differentiated breast cancer risk. https://t.co/R0TfHiNg7S
Poorly differenciated cancers are in strong correlation with defective estrogen signaling. https://t.co/2bbvQ67K6Z
The higher the ER expression in tumor cells, the stronger is the anticancer capacity of estrogen. https://t.co/9FOCAoRp6P
In tumor cells estrogen receptor may be activated by E2 and it leads to self directed tumor death. Open access. https://t.co/0qZ20KqBea
The stronger the dedifferentiation of breast cancers, the higher is the disturbance of estrogen signaling and the d https://t.co/cOsne73DPT
High estrogen level increases the ER expression of tumors and helps the self-regulated domestication and apoptotic https://t.co/464gRfoJjS
Higher risk for TNBC in young, premenopausal women is a medical mistake attributed to inadequate statistical evalua https://t.co/DyPDhY7SQW
The stronger the estrogen receptor signaling in women, the lower the risk for undifferentiated TNBC development. https://t.co/CprWalYSj7
The stronger the metabolic and hormonal imbalance in women, the higher is the risk for breast cancer. https://t.co/zWqjZnWG4J
High TNBC risk of dark skinned American women may be explained by their low light exposure in North-America, and th https://t.co/qFBYSWDNU4
The grade of defect in metabolic and hormonal equilibrium is directly associated with TNBC risk for both premenopau https://t.co/JcRUw6u3uv
TNBC risk is in close correlation with BRCA gene mutation attributed to the defective transcriptional activity of E https://t.co/RcN9t6Qus8
The higher the ER expression of tumor cells, the stronger the killing capacyty of estrogen exposure. https://t.co/7Aev5caBEb
The grade of defect in metabolic and sex hormone equilibrium is directly associated with TNBC risk for both premeno https://t.co/LVD9z6wC8T
The higher the ER expression of tumor cells, the stronger the killing capacity of estrogen exposure. https://t.co/Ocg3ce5rEM
The tumor suppressing effect of excessive and the liberating effect of defective estrogen supply has disproportiona https://t.co/tOuYlKp2cQ
The grade of defect in metabolic and hormonal equilibrium is directly associated with TNBC risk for both premenopau https://t.co/eesyLTKCe2
The tumor suppressing effect of excessive and the liberating effect of defective estrogen supply has disproportiona https://t.co/Y1mvwqotxG
The higher the ER expression of tumor cells, the stronger the killing capacyty of estrogen exposure. https://t.co/m6tYHnmU2X
The grade of defect in metabolic and hormonal equilibrium is directly associated with TNBC risk for both premenopau https://t.co/U4hAKV6cU4
The more defective the estrogen signaling the higher is the risk for triple negative breast cancer. Open access. https://t.co/rzRsmixfIy
The more defective the estrogen signaling the higher is the risk for TNBC development. https://t.co/xy6I1Dmxsf
The tumor suppressing effect of excessive and the liberating effect of defective estrogen supply has disproportiona https://t.co/znOVo53Mvt
Lack of ERs and Her2 receptors in TNBC cells blocks the genome stabilizer mechanisms by interplay between ERs and G https://t.co/5wLeROjzju
The stronger the defect of estrogen receptor signaling, the higher is the risk for TNBC development. https://t.co/TucE6gPw80
The stronger the cancer risk factor, the higher is the risk for ER-negative tumor development. https://t.co/GN5XIwinB4
In the estrogen-rich milieu of multiparous women, a fairly decreased number and percentage of ER-positive tumors is https://t.co/jxRVnRH9Je
The grade of defect in ER signaling is directly associated with poorly differentiated, ER-negative breast cancer ri https://t.co/fBEtV8zJGt
The clinical and biologic significance of ER-protein expression in breast cancers has been equivocally established. https://t.co/EsXRww1i9f
The grade of defect in estrogen signaling is directly associated with poorly differentiated, ER-negative breast can https://t.co/JEow88cSS8
ER+ and ER- breast cancers have no different or quite inverse causal factors. The stronger the cancer risk factor, https://t.co/uwHn1PcxDb
In young breast cancer cases, the higher percentage of ER- tumors may be attributed to the stronger killer capacity https://t.co/lstho0CSh4
Many investigations mistakenly propose that parity associated increased estrogen concentration may differently or e https://t.co/3MHjzemRyq
The higher the grade of insulin resistance of patients, the higher is the risk for poorly differentiated, ER-negati https://t.co/3kSfKTNPLb
Increased estrogen level in pregnancy does not affect inversely the initiation of ER+ and ER- breast cancers. High https://t.co/mfpcMI7rrG
The stronger is the defect of estrogen signaling, the higher is the risk for poorly developed, ER-negative breast c https://t.co/CAbxHLzUtx
High estrogen level in pregnancy is erroneously believed to act differently or quite inversely on the development o https://t.co/ksspRmMTKV
The clinical and biologic significance of ER-protein expression in breast cancers has been equivocally established. https://t.co/I6mS6TvKPL
The relatively higher percentage of surviving ER-negative mammary cancers among young cases may be attributed to th https://t.co/jOMpfHtq5j
Increased TNBC risk of dark skinned American women may be attributed to their light deficiency associated hormonal https://t.co/1eGM6hp6Us
In young women, the higher precentage of ER-negative tumors may be attrubuted to the stronger elimination of ER+ on https://t.co/4NjoNMXGyd
The grade of defect in estrogen signaling is directly associated with poorly differentiated, ER-negative breast can https://t.co/6P7qTKmJwY
In infertile women, the insufficient estrogen supply has a defective killing capacity, even against the predominant https://t.co/vksONasFtF
Estrogens, being the specific ligand for ERs, may preferentially block the development and progression of ER-positi https://t.co/xTQTbv3vXA
Among young cases, the relatively higher percentage of surviving ER-negative mammary cancers may be attributed to t https://t.co/vidPaELfVr
The relatively higher percentage of surviving ER-negative mammary cancers among young cases may be attributed to th https://t.co/UIeHKvc9Y3
ER+ and ER- breast cancers are not different tumors with different etiology. The milder the defect of estrogen sign https://t.co/Ovfn2WRypI
The tumor suppressing effect of excessive and the liberating effect of defective estrogen supply has disproportiona https://t.co/rE8kKd5jSn
The stronger the defect of estrogen receptor signaling, the higher is the risk for the development of poorly develo https://t.co/lQJaGNOaWJ
The tumor suppressing effect of excessive and the liberating effect of defective estrogen supply has disproportiona https://t.co/GMK0DLnipZ
The etiology of the development of ER-positive and ER-negative breast cancers is mistakenly believed to be quite di https://t.co/WEaVLpGRvl
In African-American women, the relatively high risk of developing TNBCs may be explained by the incongruence betwee https://t.co/K2F84WQAXN
The tumor suppressing effect of excessive and the liberating effect of defective estrogen supply has disproportiona https://t.co/4pB55fhkdx
The tumor suppressing effect of excessive and the liberating effect of defective estrogen supply has disproportiona https://t.co/f9qBFXuIri
The stronger the defect of estrogen signaling, the higher is the risk for poorly differentiated ER- breast cancer. https://t.co/atZeekjguO
The stronger the defect of estrogen signaling, the higher is the risk of poorly differentiated breast cancer. https://t.co/Cta01RQoXJ
Cycling estrogen level in premenopausal cases have stronger suppressive effect on ER+ breast cancers, while weaker https://t.co/NxAJLWRIIZ
In cycling young women, increased rates of pooely differentiated ER-negative breast cancers may be attributed to th https://t.co/DQAgB5aMtS
In African-American women, increased risk for poorly differentiated breast cancer, such as TNBC, is determined by e https://t.co/yaOyoPd9bI
Higher risk for TNBC in African-American women is defined by their estrogen loss and associated hormonal disorders https://t.co/YBPSbPEfpB
The grade of defect in metabolic and hormonal equilibrium is directly associated with TNBC risk for dark skinned wo https://t.co/HQ8bVNh1FY
In cycling young women, relatively higher rate of poorly differentiated ER-negative cancers may be explained by the https://t.co/RyGYHwblYF
The stronger the metabolic and hormonal disorder in women,, the higher is the risk of poorly differentiated breast https://t.co/uwcGYI6PAP
The detection of ESR1-gene amplification or restoration of ER-alpha expression predicts effective tumor response to https://t.co/I5lAFfUPsM
In African-American women, the relatively high risk of developing poorly differentiated breast cancers may be expla https://t.co/t6SQ6IODvc
Relatively high percentage of ER-negative breast cancer rate among cycling young women may be attributed to the low https://t.co/PXr44KEIwP
If estrogens would have even the slightest carcinogenic capacity, tumor birth would be a typical event instead of c https://t.co/4wq63PGx0R
During pregnancy, an extreme increase in the estrogen supply of the fetoplacental unit serves as an exquisite safeg https://t.co/Xl3WHVreih
The relatively high percentage of surviving TNBCs in young cases may be attributed to the low incidence rate of mor https://t.co/twvmX452Ap
In women, the most important preventive strategy against breast cancers – included TNBCs – is the strict control an https://t.co/RjPCMDoe3g
The grade of defect in metabolic and hormonal equilibrium is directly associated with TNBC risk for women during th https://t.co/QJKK8IucOv
ER signaling is the chief safeguard of genome stability in the strong interplay with DNA-controlling and -repairing https://t.co/lBFLdzzU0v
ER signaling is the chief safeguard of genome stability in the strong interplay with DNA-controlling and -repairing https://t.co/HbpYCkElvn
ER signaling is the chief safeguard of genome stability in the strong interplay with DNA-controlling and -repairing https://t.co/yWULcutNFK
In BRCA gene mutation carrier women, difficulties in estrogen signaling may be associated with the fairly decreased https://t.co/aQvH27pqB2