Estrogens provide the physiologic surveillance of genome stability and estrogen withdrawal from either healthy or ill patients is a major medical mistake. https://t.co/acOWkeSKmz

Upregulation of estrogen signaling displays a unique dichotomy, ensuring the survival and safe proliferative activity of privileged healthy cells, whilst inducing apoptotic death of malignant tumor cells https://t.co/SunVUKNHPY

De novo or acquired "antiestrogen resistance" of tumors may be attributed to the defective or later exhausted genetic capacity of patients for the extreme upregulation of estrogen signal https://t.co/PmEcw00J2V

Estrogens provide the physiologic surveillance of genome stability and estrogen withdrawal from either healthy or ill patients is a major medical mistake. https://t.co/FoCG0I86iD

De novo or acquired "antiestrogen resistance" of tumors may be attributed to the defective or later exhausted genetic capacity for the extreme upregulation of estrogen receptor expression and aromatase synthesis https://t.co/8UjLaHBUNk

Whatever type of available endocrine therapies may be used, including estrogen, antiestrogen treatment or oophorectomy, an extreme upregulation of estrogen signaling may lead to successful prevention and treatment for breast cancer. https://t.co/OYpNwa1FIZ

estrogen signal is the chief regulator of genomic machinery and its blockade is a serious medical mistake. https://t.co/i5lV96l4OP

Endocrine disruptors, such as antiestrogens, may achieve tumor regression in the minority of cases, as only an inte https://t.co/HzIzRSfa9Y

Endocrin disruptors may achieve tumor regression in the minority of cases, as only an intensive upregulation of est https://t.co/KG8YgMlEVw

De novo or acquired "antiestrogen resistance" of tumors may be attributed to the defective or later exhausted genet https://t.co/HiIGeR4gDE

estrogen signal is the chief regulator of genomic machinery and its blockade is a serious medical mistake. https://t.co/M06MGCyFgX

estrogen signal is the chief regulator of genomic machinery and its artificial blockade is a serious medical mistak https://t.co/gPZDx2NDeS

Estrogens provide the physiologic surveillance of genome stability and estrogen withdrawal from either healthy or i https://t.co/4gVHJcp6jB

Upregulation of estrogen signaling displays a unique dichotomy, ensuring the survival and safe proliferative activi https://t.co/v654C6LcbL

De novo or acquired "antiestrogen resistance" of tumors may be attributed to the defective or later exhausted genet https://t.co/xPtqC9uCrk

De novo or acquired "antiestrogen resistance" of tumors may be attributed to the defective or later exhausted genet https://t.co/RcisiCOtRy

Antiestrogens are effective in the minority of breast cancers by the compensatory upregulation of estrogen signalin https://t.co/SgM9u3LrNk

Antiestrogens may be effective anticancer agents only by the compensatory upregulation of estrogen signaling https://t.co/IkDc8QedLe

Estrogens provide the physiologic surveillance of genome stability and estrogen withdrawal from either healthy or i https://t.co/z2IdznWX9C

estrogen signal is the chief regulator of genomic machinery and its blockade is a serious medical mistake. https://t.co/nMTLzAQaAm

Antiestrogen treatment is a medical mistake; however, studies on molecular events in responsive and non responsive https://t.co/m4wfbNbc6Y

There is no resistance to antiestrogens in tumors; but rather non responsive tumors cannot compensate the impact of https://t.co/G758aQTTo2

Estrogen activation of ERs is the highly waited wonder capable of curing all human diseases including cancer https://t.co/sCbe0oLNdr

estrogen signal is the chief regulator of genomic machinery and its blockade is a serious medical mistake. https://t.co/qNrDpNcRvy

Upregulation of estrogen signaling displays a unique dichotomy, ensuring the survival and safe proliferative activi https://t.co/pmr1PBTHrQ

Antiestrogens may achieve tumor response only by the compensatory upregulation of estrogen signaling https://t.co/MTeLr995J7

Upregulation of estrogen signaling displays a unique dichotomy, ensuring the survival and safe proliferative activi https://t.co/qwSMyJXypx

Antiestrogen treatment of tumors is a medical mistake. Blockade of liganded ER activation may achieve tumor respons https://t.co/RvhVoutPCL

The deceiving weak anticancer capacity of antiestrogens may be attributed to the marvelous compensatory efforts of https://t.co/LBkt8TAqUv

Ineffective antiestrogen therapies are acting as catalysts for fundamental changes in erroneous therapeutic princip https://t.co/HNSxuWCcEU

Antiestrogen prevention and treatment of tumors is a dangerous medical mistake https://t.co/OyB95OzPNy

Antiestrogen use may achieve tumor response in the minority of breast cancer cases, when tumor shows a compensatory https://t.co/eMLqEwFDer

Antiestrogen blockade of ERs may induce tumor response when the compensatory ER activation is strong. https://t.co/Z9p4Svv1hg

Antiestrogens have weak, unreliable anticancer effects as they block the highest regulatory capacity of genomic mac https://t.co/CAmkK946Hz

Antiestrogens may achieve tumor response via provoking a compensatory upregulation of estrogen signaling. https://t.co/9myxPykWwg

Antiestrogen blockade of ERs may cause tumor response only via the compensatory upregulation of estrogen signaling https://t.co/ozpsnOKzpE

Antiestrogens may induce tumor response via a compensatory upregulation of estrogen receptor signaling https://t.co/YyC9bGxK7J

ER blockade of breast cancer may achieve tumor response only when tumor cells may show compensatory ER activation. https://t.co/5OusYHDqs8

Estrogen signaling is not a promoter of DNA-damage and tumor growth. but rather helps DNA repair and apoptosis. https://t.co/NmnWvNQeWh

The transient anticancer effect of antiestrogens may be attributed to the compensatory activation of ERs and DNA re https://t.co/I1IaXA3AOs

In tumors, ER blockade may cause transient regression when tumor cell show compensatory upregulation of ER signalin https://t.co/Ym0XcJNgTM

Both healthy and cancer cells fight against antiestrogens via ER activation leading to immune defense and tumor dea https://t.co/jxC7DaGUeJ

Antiestrogens may be effective only by the upregulation of estrogen signaling, the fight against estrogen is a mist https://t.co/Wdfazm9FXq

Antiestrogen treatment induces compensatory activation of ER signal in 25-30% of patients and their tumors. https://t.co/ER3xOWuVh1

Tumors unresponsive to antiestrogen treatment are not resistant, but rather incapable of upregulation of ER signali https://t.co/FnDfS5rzFE

Lack of tumor response in the majority of breast cancer cases is a proof that antiestrogen therapy is an error. https://t.co/0qMWbKl1d6

In tumors non responsive to antiestrogens,, the compensatory increased ER signaling is exhausted. https://t.co/jJBKb4z63D

Antiestrogens are the most toxic chemicals as they inhibit the main regulator of genomic machinery inducing a block https://t.co/F7r6poIfRr

In tumor cells, a compensatory increased estrogen signal may induce apoptosis instead of the liganded ER blockade. https://t.co/ItV5HTb98w

In tumor cells, a compensatory increased estrogen signal may induce apoptosis instead of the liganded ER blockade. https://t.co/1NYTgUq1DZ

Estrogen blockade is an erroneous means of cancer cure its effectivity depends on the compensatory ER activation. https://t.co/cHn0NWy6iM

Is Premarin, alone, the key to successful menopausal hormone therapy? Suba Z. https://t.co/HlbCkkEUVh

In the vast majority of antiestrogen treated tumors, ER blockade induces tumor growth instead of regression. https://t.co/vthCzTfeQZ

Prophylactic antiestrogen treatment of BC patients is a Russian rulette taking blind risk for tumor growth. https://t.co/AtqdJYcG0N

Antiestrogen compounds were developed against breast cancer based on the erroneous concept of estrogen induced canc https://t.co/mREnVp98eZ

Antiestrogen compounds were developed against breast cancer based on the erroneous concept of estrogen induced canc https://t.co/v3A4NU895Q

In endocrine responsive tumors, a compensatory increased E2 signaling helps apoptotic death instead of the blockade https://t.co/ODYJhgaBIY

In tumor cells, abundance of activated ERs helps DNA stabilization, whilst their medical blockade induces tumor gro https://t.co/ErhVGeqXxH

Blockade of liganded activation of ERs may show anticancer efficacy via compensatory upregulation of ER signaling https://t.co/k9W8NpgXGE

Antiestrogens may achieve tumor response only when the compensatory upregulation of ER activation is strong enough. https://t.co/EXNaNp47OA

Various anticancer efficacy of antiestrogens may be attributed to the compensatory upregulation of genomic machiner https://t.co/IoNQhrWgAQ

Increased estrogen receptor expression in "antiestrogen resistant" tumor cells is an insufficient counteraction aga https://t.co/DKoGRDHo47

There is no "antiestrogen resistance" but rather an uncompensated blockade of ERs leading to unrestrained tumor gro https://t.co/XDhwrxRP0V

Blockade of estrogen signal in tumors may induce regression only via a compensatory upregulation of ERs. https://t.co/akpD7GhGfn

Antiestrogens may be effective only by the compensatory upregulation of estrogen signaling https://t.co/eGNQ5Lv2Io

Antiestrogens were developed against cancer based on a concept that increased estrogen level causes damage in DNA https://t.co/TBlTLtmg5S

In antiestrogen treated tumors, upregulation of ERs is not a survival technikue, but rather drives self directed de https://t.co/WHjb3i9TPb

Tamoxifen induces the upregulation of ER expression and estrogen synthesis promoting a self directed death of tumor https://t.co/snFjGBDoh1

Estrogen receptor blockade upregulates the receptor expression leading to increased ER signaling and apoptosis in t https://t.co/x6sVkF1IAd

Blockade of liganded ER activation activates both E2 synthesis and ER expression leading to apoptosis in tumor cell https://t.co/La0sqLUwNv

Tamoxifen induced transient tumor response may be attributed to a compensatory ER activation instead of ER blockade https://t.co/XJuAHspJsj

Tamoxifen therapy of tumors is effective only when it may provoke a strong compensatory upregulation of ER signalin https://t.co/TsdUXEFBdh

Blockade of liganded ER signaling in tumors is not an appropriate therapy, but rather a medical mistake. https://t.co/UcR3I26poN

Estrogens provide the surveillance of DNA stability and its withdrawal from patients is a major medical mistake. https://t.co/sA8rnM3xCm

"antiestrogen resistance" of tumors may be attributed to the defective capacity of patient for a compensatory react https://t.co/hTREXcPRxA

Inhibition of the activation of estrogen receptors in tumors is a medical failure as it does not block proliferatio https://t.co/wCtvxtI70m

The blockade of liganded activation of ERs may be effective against cancer when ERs show compensatory upregulation. https://t.co/M351aRdfEo

TAM resistance of tumors is a misbelief, ER blockade may induce tumor response only by compensatory ER activation. https://t.co/kT5AC5gETu

Blockade of liganded ER signaling in tumor cells is a medical mistake as ERs are the chief regulators of apoptosis. https://t.co/AEgqnG5fdF

In antiestrogen treated patients, blockade and compensatory activation of ERs result in chaotic regulation of genom https://t.co/9a0V5RotLH

"Antiestrogen resistance" of tumors may be attributed to defective genetic capacity of patients for ER activation. https://t.co/Li8oT7bCyF

"Antiestrogen resistance" of tumors may be a defective capacity of patients for the upregulation of estrogen signal https://t.co/YJUIpUZuvV

Antiestrogen compounds were developed against breast cancer based on the erroneous concept that increased estrogen https://t.co/vPPYWmKGLS

An extreme compensatory upregulation of estrogen signaling may lead to successful prevention and treatment for brea https://t.co/giHXDQSIjv

Antiestrogen compounds were developed against breast cancer based on the erroneous concept that increased estrogen https://t.co/dARAPmOt96

Antiestrogens may be effective only by the upregulation of estrogen signaling https://t.co/7Fzgiz8Shn

Estrogen blockade may induce tumor response only by the compensatory upregulation of estrogen signaling https://t.co/1n6V4sVrZU

Upregulation of estrogen signaling displays a unique dichotomy, ensuring the survival of privileged healthy cells, https://t.co/HCqGJY71EI

Upregulation of estrogen signaling ensures the apoptotic death of malignant tumor cells. https://t.co/eQoVdiIXCX

Antiestrogens may be effective only by the upregulation of estrogen signaling https://t.co/QeVNNwCkT2

Antiestrogens may be effective only by the compensatory upregulation of estrogen signaling https://t.co/UVSsvZ04uG

Antiestrogens may be effective only by the upregulation of estrogen signaling https://t.co/qys8NMCnxt

Abrupt withdrawal of crucial mediators, like estrogen, induces unpredictably strong compensatory mechanisms for the https://t.co/MJv8tG5Huj

antiestrogen treatment may not accomplish a direct downregulation of cell proliferation, but rather it provokes a c https://t.co/1FzuPAuMUe

Antiestrogens may be effective only by the upregulation of estrogen signaling https://t.co/EuvUBSCb8K

Antiestrogens provoke tumor response only by the compensatory upregulation of ER signaling. https://t.co/j5SlbeGn5D

Antiestrogens may be effective only by the compensatory upregulation of estrogen signaling https://t.co/Iouj13Drdy

Upregulation of estrogen signaling induces apoptotic death of malignant tumor cells. https://t.co/FWHdwDzv6L

Antiestrogens may be effective only by the compensatory upregulation of estrogen signaling https://t.co/IcgPDRWBg4

Antiestrogen compounds were developed against breast cancer based on the erroneous concept that increased estrogen https://t.co/XFvKE5WLk4