NGF drives inflammation and contributes to peripheral sensitisation (eg exogenous NGF causes hyperalgesia) but it's significance depends on cause of pain - i.e. antiNGF mAbs are unlikely to be a broad-spectrum analgesics. More in this review: https://t.co/
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NGFは炎症性傷害に関連する細胞から放出され、侵害受容器末端の様々なイオンチャネル・受容体の活性化促進。短時間で神経脱分極と感作を促進し、また長時間でNGF/TrkA複合体は神経逆行輸送され、DRG内のNGF/TrkAシグナリングが遺伝子発現。#338 https://t.co/37OLZBI6Px https://t.co/7gwjED5MuM